Kimberly Byrnes, PhD

Kimberly Byrnes, PhD

Name: Kimberly Byrnes, PhD

USU Department of Primary Appointment: 
Anatomy, Physiology and Genetics
Faculty Rank: 
Associate Professor
Location: 
Uniformed Services University of the Health Sciences, Bethesda, MD

Research Interests:
Brain and spinal cord trauma
Inflammation

Office Phone: 
(301) 295-3217

Education

Postdoctoral Fellowship 2004-2007 Georgetown University
Postdoctoral Fellowship 2003-2004 Uniformed Services University
PhD 2003 Uniformed Services University
BS 1997 University of Pittsburgh

Representative publications, projects, and/or deployments

  • Associate Professor with Tenure, Uniformed Services University 2014
  • Assistant Professor, Uniformed Services University 2009
  • Research Assistant Professor, Georgetown University 2007
  • Von Leden RE, Khayrullina G, Moritz KE, Byrnes KR. Age exacerbates microglial activation, oxidative stress, inflammatory and NOX2 gene expression, and delays functional recovery in a rodent model of spinal cord injury. J Neuroinflammation; 2017, In Press.
  • Brabazon F, Wilson CM, Jaiswal S, Frey WH II, Byrnes KR. Intranasal insulin treatment of an experimental model of moderate traumatic brain injury. J Cerebral Blood Flow Metab, 2017. In Press.
  • Brabazon F, Wilson CM, Shukla DK, Mathur S, Jaiswal S, Bermudez S, Byrnes KR, Selwyn R. [18F]FDG-PET combined with diffusion MRI enhances the detection of traumatic brain injury in rats. J Neurotrauma. 2017; 34(5):1074-1085.
  • Von Leden RE, Yauger YJ, Khayrullina G, Byrnes KR. Central Nervous System Injury and NADPH Oxidase: Oxidative Stress and Therapeutic Targets. J Neurotrauma, 2017; 34(4):755-764.
  • von Leden RE, Selwyn RG, Jaiswal S, Wilson CM, Khayrullina G, Byrnes KR. 18F-FDG PET imaging of injured rat spinal cord reveals depressed glucose uptake correlating with lesion volume and functional recovery. Neurosci Letters. 2016; 621:126-32.
  • Selwyn RG, Cooney SJ, Khayrullina G, Hockenbury N, Wilson CM, Jaiswal S, Bermudez S, Armstrong RC, Byrnes KR. Outcome after repetitive mild traumatic brain injury is temporally related to glucose uptake profile at time of second injury. J Neurotrauma. 2016; 33:1479-91.
  • Khayrullina G, Bermudez S, Byrnes KR. Inhibition of NOX2 reduces locomotor impairment, inflammation and oxidative stress after spinal cord injury. J Neuroinflammation. 2015, 12:172.
  • Cooney SJ, Bermudez-Sabogal SL, Byrnes KR. Cellular and temporal expression of NADPH oxidase (NOX) isotypes after brain injury. J Neuroinflammation, 2013 10:155.