Peter J Klover

M.S., Ph.D.

Department of Primary Appointment:
School of Medicine
Staff Scientist
Location: Uniformed Services University of the Health Sciences, Bethesda, MD
Research Interests:
tuberous sclerosis complex
Skin regeneration
Office Phone


Doctor of Philosophy in Biochemistry and Biophysics (2005) University of Rochester
Master of Science in Biochemistry and Biophysics (2002) University of Rochester
Bachelor of Science in Microbiology (1997) Washington State University


Klover PJ, Thangapazham RL, Kato J, Wang JA, Anderson SA, Hoffmann V, Steagall WK, Li S., McCart E, Nathan N, Bernstock JD, Wilkerson MD, Dalgard CL, Moss J, Darling TN. (2017) Tsc2 disruption in mesenchymal progenitors results in tumors with vascular anomalies overexpressing Lgals3. eLife. Accepted Manuscript (in press).

Thangapazham RL, Klover P, Li S, Wang JA, Sperling L, Darling TN. (2014) A model system to analyse the ability of human keratinocytes to form hair follicles. Exp Dermatol. Jun;23(6):443-6.

Thangapazham RL, Klover P, Wang JA, Zheng Y, Devine A, Li S, Sperling L, Cotsarelis G, Darling TN. (2014) Dissociated human dermal papilla cells induce hair follicle neogenesis in grafted dermal-epidermal composites. J Invest. Dermatol. Feb 134(2) 538-40.

Klover, P.J., Muller, W., Robinson, G., Yamaji D, Pfeiffer, R., and Hennighausen L. (2010). Loss of STAT1 from mouse mammary epithelium results in an increased neu-induced tumor burden. Neoplasia, 12(11) 899-905.

Klover, P., Chen, W., Zhu, B. M., and Hennighausen, L. (2009) Skeletal muscle growth and fiber composition in mice are regulated through the transcription factors STAT5A/5B: linking growth hormone to the androgen receptor. FASEB J 23, 3140-3148.

Klover, P., and Hennighausen, L. (2007) Postnatal body growth is dependent on the transcription factors signal transducers and activators of transcription 5a/b in muscle: a role for autocrine/paracrine insulin-like growth factor I. Endocrinology 148, 1489-1497.

Klover, P. J., Clementi, A. H., and Mooney, R. A. (2005) Interleukin-6 depletion selectively improves hepatic insulin action in obesity. Endocrinology 146, 3417-3427.